Keywords: سمیت گلوکسیت; Pancreatic islets; Fatty acids; Glucolipotoxicity; Acetylcholine; Insulin secretion;
مقالات ISI سمیت گلوکسیت (ترجمه نشده)
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Keywords: سمیت گلوکسیت; CNS; central nervous system; INS-1; insulin secreting β-cell line originated from a rat insulinoma; GK rat; Goto-Kakizaki rat; NOD mouse; non-obese diabetic mouse; TKP; tryptophan/kynurenine pathway; Trp; tryptophan; KYN; kynurenine; 3HK; 3-OH-kynureni
CD36 mediates lipid accumulation in pancreatic beta cells under the duress of glucolipotoxic conditions: Novel roles of lysine deacetylases
Keywords: سمیت گلوکسیت; CD36; Caspase 3; Glucolipotoxicity; Histone deacetylases; Pancreatic beta cells;
Sirtuin 5 overexpression attenuates glucolipotoxicity-induced pancreatic β cells apoptosis and dysfunction
Keywords: سمیت گلوکسیت; SIRT5; β cell; Glucolipotoxicity; Apoptosis; Insulin secretion;
HAMSCs/HBMSCs coculture system ameliorates osteogenesis and angiogenesis against glucolipotoxicity
Keywords: سمیت گلوکسیت; Human amnion-derived mesenchymal stem cells; Human bone marrow mesenchymal stem cells; Glucolipotoxicity; Osteogenesis; Angiogenesis; DM; diabetes mellitus; HAMSCs; human amnion-derived mesenchymal stem cells; HBMSCs; human bone marrow mesenchymal stem ce
Egr-1 transactivates WNT5A gene expression to inhibit glucose-induced β-cell proliferation
Keywords: سمیت گلوکسیت; β-cell; Wnt5a; Egr-1; Glucolipotoxicity; Lipotoxicity; Saturated fatty acids;
Glycerol-3-phosphate phosphatase/PGP: Role in intermediary metabolism and target for cardiometabolic diseases
Keywords: سمیت گلوکسیت; Glycerol-3-phosphate phosphatase; Phosphoglycolate phosphatase; Glycerolipid/fatty acid cycle; HAD-like phosphatase; Metabolic syndrome; Type 2 diabetes; AUM; Aspartate-based, ubiquitous, Mg2+-dependent phosphatase; DAG; di-acylglycerol; Glptx; glucolipot
Glucolipotoxicity diminishes cardiomyocyte TFEB and inhibits lysosomal autophagy during obesity and diabetes
Keywords: سمیت گلوکسیت; ATGL; Adipose triglyceride lipase; CHOP; CCAAT-enhancer-binding protein homologous protein; CLEAR; coordinated lysosomal enhancement and regulation; CMA; chaperone mediated autophagy; CQ; chloroquine; eIF2α; eukaryotic translation initiation factor 2α;
Mitochondrial uncoupling protein-2 is not involved in palmitate-induced impairment of glucose-stimulated insulin secretion in INS-1E insulinoma cells and is not needed for the amplification of insulin release
Keywords: سمیت گلوکسیت; BSA, bovine serum albumin; DAPI, 4′,6-diamidino-2-phenyl-indole; GSIS, glucose-stimulated insulin secretion; UCP, uncoupling protein; NEFA, non-esterified fatty acidPancreatic beta cells; Glucolipotoxicity; Uncoupling protein-2 (UCP2); Glucose-stimulated
Uncoupling protein-2 attenuates palmitoleate protection against the cytotoxic production of mitochondrial reactive oxygen species in INS-1E insulinoma cells
Keywords: سمیت گلوکسیت; Pancreatic beta cells; Glucolipotoxicity; Cytoprotection; Mitochondrial dysfunction; Uncoupling protein-2 (UCP2); Reactive oxygen species; INS-1E insulinoma cells; Non-esterified fatty acids; Obesity; Type 2 diabetes; BSA; bovine serum albumin; DAPI; 4â²
Natural history of β-cell adaptation and failure in type 2 diabetes
Keywords: سمیت گلوکسیت; β-cell failure; β-cell development; β-cell programming; Glucolipotoxicity; Islet biology; Insulin resistance
Toxicity generated through inhibition of pyruvate carboxylase and carnitine palmitoyl transferase-1 is similar to high glucose/palmitate-induced glucolipotoxicity in INS-1 beta cells
Keywords: سمیت گلوکسیت; AMPK; AMP-activated protein kinase; CHOP; C/EBP homologous protein; CPT-1; Carnitine palmitate transferase-1; DAG; diacylglycerol; ER; endoplasmic reticulum; Eto; etomoxir; FAO; fatty acid oxidation; HG/PA; high glucose/palmitate; JNK; C-Jun N-terminal ki
Elevated toll-like receptor 3 inhibits pancreatic β-cell proliferation through G1 phase cell cycle arrest
Keywords: سمیت گلوکسیت; β-cell; Glucolipotoxicity; TLR3; Proliferation; Arrest;
Mediation of glucolipotoxicity in INS-1 rat insulinoma cells by small heterodimer partner interacting leucine zipper protein (SMILE)
Keywords: سمیت گلوکسیت; Glucolipotoxicity; High glucose; Palmitate; SMILE; Beta cell dysfunction;
A compound (DW1182v) protecting high glucose/palmitate-induced glucolipotoxicity to INS-1 beta cells preserves islet integrity and improves hyperglycemia in obese db/db mouse
Keywords: سمیت گلوکسیت; CHOP; ER stress; Glucolipotoxicity; INS-1 beta cell; JNK; Type 2 diabetes
Biochemical Basis and Clinical Consequences of Glucolipotoxicity
Keywords: سمیت گلوکسیت; Glucolipotoxicity; Pancreatic β-cell dysfunction; Insulin resistance;
Glycerolipid/free fatty acid cycle and islet β-cell function in health, obesity and diabetes
Keywords: سمیت گلوکسیت; Insulin secretion; Glycerolipid/fatty acid cycle; Glucolipotoxicity; Lipogenesis; Lipolysis; ABHD6; α/β-domain containing hydrolase-6; ADRP; adipocyte differentiation-related protein; AGPAT; 1-acyl-sn-glycerol-3-phosphate acyltransferase; AMPK; AMP acti
Supplement of TCA cycle intermediates protects against high glucose/palmitate-induced INS-1 beta cell death
Keywords: سمیت گلوکسیت; Anaplerosis; Beta cell; Glucolipotoxicity; Mitochondria; TCA cycle;
Glucolipotoxicity in INS-1E cells is counteracted by carnitine palmitoyltransferase 1 over-expression
Keywords: سمیت گلوکسیت; Type 2 diabetes; Glucolipotoxicity; INS-1E; Palmitate; Carnitine palmitoyltransferase 1; ER-stress; GSIS; Apoptosis; p-eIF2α; CHOP/GADD153
Glucose-induced lipogenesis in pancreatic β-cells is dependent on SREBP-1
Keywords: سمیت گلوکسیت; INS-1E cells; Transduction; Adenovirus; Dominant negative SREBP; Lipid accumulation; Glucolipotoxicity;