
Exposure of any of two proapoptotic domains of presenilin 1-associated protein/mitochondrial carrier homolog 1 on the surface of mitochondria is sufficient for induction of apoptosis in a Bax/Bak-independent manner
Keywords: غشای خارجی; PSAP/Mtch1; Mimp/Mtch2; Apoptosis; Mitochondria; Outer membrane; Permeability transition; Bax; Bak; Presenilin; Alzheimer