The role of elevated autophagy on the synaptic plasticity impairment caused by CdSe/ZnS quantum dots
Keywords: Autophagy; LC3-I/II; QDs; Synaptic plasticity; Synapsin-I; Synapse density; QD; quantum dot; LC3; light chain protein 3; PE; phosphatidylethanolamine; mTOR; mammalian target of rapamycin; LTP; long-term potentiation; I/O; input/output; PPF; paired-pulse f