Biomarkers and energy reserves in the isopod Porcellionides pruinosus: The effects of long-term exposure to dimethoate

The results obtained showed that dimethoate causes toxicity by several mechanisms. This study found evidence for the inhibition of the acetylcholinesterase enzyme, which has been previously reported, and also evidence of oxidative stress, which altered the levels of GST, CAT or LPO. In addition, the study showed that the two concentrations used of dimethoate caused the activation of different general detoxification mechanisms, and also that the same concentration at different temperatures induced different toxicity responses.