Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca2 + currents and promote proarrhythmic behavior in ventricular myocytes

Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca2 + currents and promote proarrhythmic behavior in ventricular myocytes

Database: Elsevier - ScienceDirect (ساینس دایرکت)
Journal: Journal of Molecular and Cellular Cardiology - Volume 74, September 2014, Pages 115-124

Worawan B. Limpitikul, Ivy E. Dick, Rosy Joshi-Mukherjee, Michael T. Overgaard, Alfred L. Jr., David T. Yue,