Effects of Different Degrees of Sympathetic Antagonism on Cytokine Network in Patients With Ischemic Dilated Cardiomyopathy

Our results support the hypothesis that a more complete block of sympathetic activity by carvedilol induces a greater decrease in the circulating levels of proinflammatory cytokines that could explain, at least in part, the better improvement in the left ventricular remodelling and systolic function in patients with IDC.