کد مقاله | کد نشریه | سال انتشار | مقاله انگلیسی | نسخه تمام متن |
---|---|---|---|---|
929592 | 1474414 | 2014 | 8 صفحه PDF | دانلود رایگان |
• Obesity related impairments of cardiovascular functioning were assessed
• Two tasks (physical and mental) were used to evaluate cardiovascular impairments
• Obese participants exhibit blunted reactivity to the MA task
• The relationship between reactivity and obesity may be moderated by stress
• Greater adiposity predicted delayed heart rate recovery following both tasks
The role that excess adipose tissue plays in chronic inflammation gives rise to its importance as an independent risk factor in cardiovascular dysfunction. Operationalizing chronic stress as obesity, we sought to explore the relationship between obesity, perceived stress and cardiovascular reactivity and recovery from laboratory stressors.Cardiovascular function was assessed using blood pressure and heart rate. Two stress tasks (mental arithmetic and cold pressor) were employed to examine potential differences between type of stress and cardiovascular response. Body mass index (BMI) was able to predict dysfunction in both cardiovascular reactivity and recovery. Participants with a higher BMI exhibited blunted systolic blood pressure and heart rate reactivity to the mental arithmetic task. In contrast, BMI has an incongruent effect on blood pressure reactivity to the cold pressor task that is dependent on the level of perceived stress. This suggests that in some instances the effect of BMI on cardiovascular response to acute stress may be moderated by perceived stress. Further, we found greater adiposity was related to delayed heart rate recovery following both stress tasks.
Journal: International Journal of Psychophysiology - Volume 94, Issue 1, October 2014, Pages 1–8