Keywords: حرکات غیر دائمی غیر طبیعی; Abnormal involuntary movements; Psychosis risk; Children; Adolescents; Epidemiology;
مقالات ISI حرکات غیر دائمی غیر طبیعی (ترجمه نشده)
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Keywords: حرکات غیر دائمی غیر طبیعی; 5-HIAA; 5-hydroxyindolacetic acid; 5-HT; serotonin; 6-OHDA; 6-hydroxydopamine; AIMs; Abnormal involuntary movements; ARC; activity-regulated cytoskeletal-associated protein; DA; dopamine; DOPAC; 3,4-dihydroxyphenylacetic acid; EDTA; ethylenediaminetetraac
Keywords: حرکات غیر دائمی غیر طبیعی; Schizophrenia; Neurological soft signs; Abnormal involuntary movements; Catatonic symptoms; Neuroimaging;
Regulation of Pleiotrophin and Fyn in the striatum of rats undergoing L-DOPA-induced dyskinesia
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; DA; dopamine; L-DOPA; L-3,4-dihidroxiphenyl-alanine; LID; L-DOPA induced dyskinesia; MSNs; medium spiny neurons; NMDA; N-methyl-d-aspartate; NMDAR; NMDA receptor; PD; Parkinson's Disease;
Selegiline increases on time without exacerbation of dyskinesia in 6-hydroxydopamine-lesioned rats displaying l-Dopa-induced wearing-off and abnormal involuntary movements
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; ALO; axial, forelimb and orolingual; ANOVA; analysis of variance; Arc; activity-regulated cytoskeleton-associated protein; CDS; continuous dopaminergic stimulation; COMT; catechol-O-methyltr
Behavioral and cellular dopamine D1 and D3 receptor-mediated synergy: Implications for L-DOPA-induced dyskinesia
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson's disease; L-DOPA-Induced dyskinesia; Abnormal involuntary movements; D1R-D3R interactions; DA; Dopamine; DOPAC; Dihydroxyphenylacetic acid; PD; Parkinson's disease; LID; L-DOPA-Induced Dyskinesia; D1R; Dopamine D1 Receptor; D2R; Dopamine D2 Rec
mGlu4 allosteric modulation for treating Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson's disease; Dyskinesia; Glutamate; Dopamine; mGlu4; 6-OHDA; 6-hydroxydopamine; ADX88178; 4-methyl-N-[5-methyl-4-(1H-pyrazol-4-yl)-1,3-thiazol-2-yl]pyrimidin-2-amine; AIM; abnormal involuntary movements; CODA-RET; complemented donor acceptor reson
Genetic deletion of Rhes or pharmacological blockade of mTORC1 prevent striato-nigral neurons activation in levodopa-induced dyskinesia
Keywords: حرکات غیر دائمی غیر طبیعی; AIMs; abnormal involuntary movements; DA; dopamine; DARPP-32; dopamine and cAMP-regulated phosphoprotein, Mr 32Â kDa; D1R; D1 receptor; D2R; D2 receptor; ERK; extracellular-regulated kinase; L-DOPA; levodopa; LID; levodopa-induced dyskinesia; MSNs; medium
In vivo characterization of a novel dopamine D3 receptor agonist to treat motor symptoms of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; Agonist; Abnormal involuntary movements; Adjuvant therapy; Dopamine D3 receptor; l-dopa induced dyskinesia; Optimal therapy; Parkinson's disease; Pharmacokinetics; Stepping test;
Effects of L-dopa priming on cortical high beta and high gamma oscillatory activity in a rodent model of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; 5-HT; serotonin; 6-OHDA; 6-hydroxydopamine; 8-OH-DPAT; (+)-8-hydroxy-2-(dipropylamino)tetralin hydrobromide; AIMs; abnormal involuntary movements; ALO; axial, limb, and orolingual; benserazide; dl-serine 2-(2,3,4-trihydroxybenzyl) hydrazide hydrochloride;
Optogenetic activation of striatal cholinergic interneurons regulates L-dopa-induced dyskinesias
Keywords: حرکات غیر دائمی غیر طبیعی; AAV; adeno-associated virus; ACh; acetylcholine; AIMs; abnormal involuntary movements; ANOVA; analysis of variance; LIDs; L-dopa-induced dyskinesias; ChAT; choline acetyltransferase; ChR2; channel rhodopsin 2; DAT; dopamine transporter; ERK; extracellular
L-DOPA-induced dyskinesia, is striatal dopamine depletion a requisite?
Keywords: حرکات غیر دائمی غیر طبیعی; L-DOPA; Dyskinesia; Abnormal involuntary movements; Non-Parkinsonian state; Striatum; Dopamine
Effect of serotonin transporter blockade on L-DOPA-induced dyskinesia in animal models of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; L-DOPA; L-3,4-dihydroxyphenylalanine; LID; L-DOPA-induced dyskinesia; MPTP; 1-methyl-4-phenyl 1,2,3,6-tetrahydropyridine; PD; Parkinson's disease; SERT; serotonin transporter; SSRIs; selec
Loss of glutamic acid decarboxylase (Gad67) in striatal neurons expressing the Drdr1a dopamine receptor prevents l-DOPA-induced dyskinesia in 6-hydroxydopamine-lesioned mice
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIM; abnormal involuntary movements; ANOVA; Analysis of variance; Gad; glutamic acid decarboxylase; l-DOPA; levodopa; LID; l-DOPA-induced dyskinesia; GABA; 6-hydroxydopamine; levodopa; dyskinesia; striatum; striatonigral;
Nitric oxide synthase inhibition decreases l-DOPA-induced dyskinesia and the expression of striatal molecular markers in Pitx3â/â aphakia mice
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson disease; l-DOPA; Abnormal involuntary movements; Nitrergic system; NO/cGMP; Pitx3 deficient aphakia mice; FosB; Histone3; Molsidomine; Zaprinast; Striatum;
Evidence for a role for α6â nAChRs in l-dopa-induced dyskinesias using parkinsonian α6â nAChR gain-of-function mice
Keywords: حرکات غیر دائمی غیر طبیعی; α-CtxMII; α-conotoxinMII; â; denotes the possible presence of other subunits in the receptor complex; 125I-RTI-121; 125I-3β-(4-iodophenyl)tropane-2β-carboxylic acid isopropyl ester; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; BS
Activation of DREAM (Downstream Regulatory Element Antagonistic Modulator), a Calcium-Binding Protein, Reduces L-DOPA-Induced Dyskinesias in Mice
Keywords: حرکات غیر دائمی غیر طبیعی; Abnormal involuntary movements; Dopaminergic denervation; Dynorphin-B; FosB; Parkinson's disease; Phospho-GluR1;
Pharmacological modulation of abnormal involuntary DOI-induced head twitch response movements in male DBA/2J mice: II. Effects of D3 dopamine receptor selective compounds
Keywords: حرکات غیر دائمی غیر طبیعی; Head twitch response; DOI; D3 dopamine receptors; 5-HT2 receptor agonist; Hallucinogenesis; Antipsychotic; AIM; abnormal involuntary movements; DOI; 2,5-Dimethoxy-4-iodoamphetamine; EPS; extrapyramidal symptoms; HTR; head twitch response; TS; Tourette Syn
Contribution of brain serotonin subtype 1B receptors in levodopa-induced motor complications
Keywords: حرکات غیر دائمی غیر طبیعی; 5-HT1B serotonin receptor; Parkinson's disease; l-DOPA-induced dyskinesia; Human; MPTP-lesioned monkey; AMPA; α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid; AIMs; abnormal involuntary movements; DA; dopamine; DL; dorsolateral; DM; dorsomedial; GAB
Striatal tyrosine hydroxylase-positive neurons are associated with l-DOPA-induced dyskinesia in hemiparkinsonian mice
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AADC; aromatic acid decarboxylase; AIMs; abnormal involuntary movements; DA; dopamine; L-DOPA; L-3,4-dihydroxyphenylalanine; LID; L-DOPA-induced dyskinesia; MFB; medial forebrain bundle; NHS; normal horse serum; OD; optical dens
Short communicationOverexpression of the dopamine D3 receptor in the rat dorsal striatum induces dyskinetic behaviors
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson's disease; Levodopa-induced dyskinesia; Dopamine receptor agonist; Tolerance; Adeno-associated virus; PD; Parkinson's disease; LID; levodopa-induced dyskinesia; AIMs; abnormal involuntary movements; EGFP; enhanced green fluorescent protein; l-DO
RGS4 is involved in the generation of abnormal involuntary movements in the unilateral 6-OHDA-lesioned rat model of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; GPCR; G-protein coupled receptor; LID; l-DOPA-induced dyskinesia; PD; Parkinson's disease; PPE-B; pre-proenkephalin B; RGS; regulators of G-protein signalling; RGS4; Parkinson's disease; l-D
Research reportEffects of noradrenergic denervation by anti-DBH-saporin on behavioral responsivity to l-DOPA in the hemi-parkinsonian rat
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; α2R; α2-noradrenergic receptor; αDBH; anti-dopamine-beta-hydroxylase saporin; ABC; avidin-biotin-peroxidase complex; AIMs; abnormal involuntary movements; ALO; axial, forelimb, orolingual; DA; dopamine; FAS; forepaw adjus
l-DOPA disrupts adenosine A2A-cannabinoid CB1-dopamine D2 receptor heteromer cross-talk in the striatum of hemiparkinsonian rats: Biochemical and behavioral studies
Keywords: حرکات غیر دائمی غیر طبیعی; AIMs; abnormal involuntary movements; BG; basal ganglia; BRET; bioluminescence resonance energy transfer techniques; FRET; fluorescence resonance energy transfer techniques; GABA; gamma-amino-butyric acid; HPD-rats; hemiparkinsonian rats (unilaterally 6-h
Angiotensin type 1 receptor blockage reduces l-dopa-induced dyskinesia in the 6-OHDA model of Parkinson's disease. Involvement of vascular endothelial growth factor and interleukin-1β
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AII; angiotensin; AIMs; abnormal involuntary movements; AT1; angiotensin type 1 receptor; BBB; blood-brain barrier; DA; dopamine; DIV; days in vitro; dNTP; deoxynucleotide triphosphate; ERK; extracellular-regulated kinase; GAD
Abnormal involuntary movement (AIM) expression following D2 dopamine agonist challenge is determined by the nature of prior dopamine receptor stimulation (priming) in 6-hydroxydopamine lesioned rats
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; Apo; apomorphine; i.p.; intraperitoneal; l-Dopa; l-3,4-dihydroxyphenylalanine; MPTP; 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine; PD; Parkinson's Disease; SKF; SKF38393; TH; tyrosine hydrox
Research ReportEffects of 5-HT1A receptor stimulation on striatal and cortical M1 pERK induction by l-DOPA and a D1 receptor agonist in a rat model of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; serotonin; 5-HT; serotonin 1A receptor; 5-HT1AR; 6-hydroxydopamine; 6-OHDA; abnormal involuntary movements; AIMs; D1 receptor; D1R; dopamine; DA; ±8-OH-DPAT; DPAT; extracellular-regulated kinase; ERK; l-DOPA-induced dyskinesia; LID; primary motor cortex;
In vivo evidence for a differential contribution of striatal and nigral D1 and D2 receptors to l-DOPA induced dyskinesia and the accompanying surge of nigral amino acid levels
Keywords: حرکات غیر دائمی غیر طبیعی; AIMs; abnormal involuntary movements; ALO; axial, limb and orolingual; DA; dopamine; DLS; dorsolateral striatum; GLU; glutamate; GP; globus pallidus; LID; l-DOPA-induced dyskinesia; 6-OHDA; 6-hydroxydopamine; SNr; substantia nigra reticulata; Dyskinesia;
Impact of L-DOPA treatment on regional cerebral blood flow and metabolism in the basal ganglia in a rat model of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; AIB; [14C]-aminoisobutyric acid; AIMs; abnormal involuntary movements; BBB; blood-brain barrier; DA; dopamine; 2-DG; [14C]-2-deoxyglucose; IAP; [14C]-iodoantipyrine; KPBS-T; potassium phosphate buffered saline containing 0.25% Triton-X; LID; L-DOPA-indu
Effects of L-DOPA and STN-HFS dyskinesiogenic treatments on NR2B regulation in basal ganglia in the rat model of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMS; abnormal involuntary movements; DA; dopamine; EP; entopeduncular nucleus; L-DOPA; l-3,4-dihydroxyphenylalanine; LID; L-DOPA-induced dyskinesia; NMDA; N-methyl-d-aspartate; PD; Parkinson's disease; SNc; substantia nigra par
Influence of corticostriatal δ-opioid receptors on abnormal involuntary movements induced by L-DOPA in hemiparkinsonian rats
Keywords: حرکات غیر دائمی غیر طبیعی; AIMs; abnormal involuntary movements; DPDPE; [d-Pen2, d-Pen5]-enkephalin; EDTA; ethylenediaminetetraacetic acid; 6-OHDA; hydroxydopamine hydrochloride, HPLC, high performance liquid chromatography; l-DOPA; l-3,4-dihydroxyphenylalanine + benserazide; LID
Animal models of l-DOPA–induced dyskinesia: an update on the current options
Keywords: حرکات غیر دائمی غیر طبیعی; l-DOPA-induced dyskinesia; 6-OHDA; MPTP; animal models; abnormal involuntary movements; Parkinson's diseaseAIMs, abnormal involuntary movements; LID, l-DOPA-induced dyskinesia; MPTP, 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine; MFB, medial forebrain bund
Glutamatergic mechanisms in the dyskinesias induced by pharmacological dopamine replacement and deep brain stimulation for the treatment of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; 6-OHDA; 6-hydroxydopamine; AIMs; abnormal involuntary movements; AMPARs; AMPA receptors; DA; dopamine; DBS; deep brain stimulation; EP; entopeduncular nucleus; GPe; external segment of the globus pallidus; GLT1; glial glutamate/aspartate transporter 1; GP
Context-driven changes in l-DOPA-induced behaviours in the 6-OHDA lesioned rat
Keywords: حرکات غیر دائمی غیر طبیعی; Context-dependent conditioning; Context-specific sensitisation; l-DOPA-induced dyskinesia; Abnormal involuntary movements; Rotation
Extent of pre-operative L-DOPA-induced dyskinesia predicts the severity of graft-induced dyskinesia after fetal dopamine cell transplantation
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson's disease; 6-OHDA; Motor behavior; Graft-induced side-effects; Abnormal involuntary movements; AIM;
Involvement of canonical and non-canonical D1 dopamine receptor signalling pathways in L-dopa-induced dyskinesia
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson's disease; L-dopa; Dopamine D1 receptor; Sensitisation; Abnormal involuntary movements; TraffickingAC, adenylyl cyclase; AIMs, abnormal involuntary movements; AMPA, α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid; cAMP, cyclic adenosine 3′,
Dopamine receptors and l-dopa-induced dyskinesia
Keywords: حرکات غیر دائمی غیر طبیعی; Parkinson's disease; l-dopa; Signalling cascade; Dopamine D1 receptor; Sensitization; Abnormal involuntary movements; Trafficking6-OHDA, 6-hydroxydopamine; AC, adenylyl cyclase; AIMs, abnormal involuntary movements; cAMP, cyclic adenosine 3′,5′-monophosph
Oral creatine supplementation attenuates L-DOPA-induced dyskinesia in 6-hydroxydopamine-lesioned rats
Keywords: حرکات غیر دائمی غیر طبیعی; Levodopa; Striatum; Abnormal involuntary movements; Parkinson’s disease; Basal ganglia; Energy deficit
Research ReportAbnormal involuntary movements (AIMs) following pulsatile dopaminergic stimulation: Severe deterioration and morphological correlates following the loss of locus coeruleus neurons
Keywords: حرکات غیر دائمی غیر طبیعی; AIMs; abnormal involuntary movements; DA; dopamine; NA; noradrenaline; DMI; desmethylimipramine; l-DOPA; l-3,4-dihydroxyphenylalanine; DSP-4; N-(-2-chloroethyl)-N-ethyl-2-bromobenzylamine; MFB; medial forebrain bundle; 6-OHDA; 6-hydroxydopamine; Dyskinesi
Effects of group I metabotropic glutamate receptors blockade in experimental models of Parkinson's disease
Keywords: حرکات غیر دائمی غیر طبیعی; Abnormal involuntary movements; In vivo; Haloperidol; Catalepsy; Turning