Deletion in the N-terminal half of olfactomedin 1 modifies its interaction with synaptic proteins and causes brain dystrophy and abnormal behavior in mice
Keywords: سیگنالینگ سلولی; MRI; Magnetic resonance imaging; Olfm1; Olfactomedin 1; Olfm1-KO; Olfm1 knockout; Q-PCR; Quantitative RT-PCR; Olfactomedin 1; AMPA receptor; Neurobiology; Proteomics; Cell signaling; Mouse; Anxiety behavior; Olfactory defects;