
Ras Promotes Growth by Alternative Splicing-Mediated Inactivation of the KLF6 Tumor Suppressor in Hepatocellular Carcinoma
Keywords: عامل رشد اپیدرمی; EGF; epidermal growth factor; ERK; extracellular signal-regulated kinase; FTS; farnesylthiosalicylic acid; KLF6; Krüppel-like factor 6; KLF6Full; full-length Krüppel-like factor 6; PCR; polymerase chain reaction; siRNA; small interfering RNA; SV1; Krüp