Computational biology analysis of platelet signaling reveals roles of feedbacks through phospholipase C and inositol 1,4,5-trisphosphate 3-kinase in controlling amplitude and duration of calcium oscillations
Keywords: IP3، inositol 1،4،5-trisphosphate; Blood platelet; Calcium signaling; Positive feedback; Negative feedbackPLC, phospholipase С; IP3, inositol 1,4,5-trisphosphate; IP3K, inositol 1,4,5-trisphosphate 3-kinase; DTS, dense tubular system; PS, phosphatidylserine; PAR, protease-activated recepto