PPARγ enhances IFNγ-mediated transcription and rescues the TGFβ antagonism by stimulating CIITA in vascular smooth muscle cells
Keywords: تخریب پروتئین; PPARγ; IFN-γ; TGF-β; CIITA; MHC II; Collagen type I; Transcriptional regulation; Protein degradation; Atherogenesis