ARF GTPases control phenotypic switching of vascular smooth muscle cells through the regulation of actin function and actin dependent gene expression
Keywords: سلول های عضلانی صاف عروقی; ARF; ADP-ribosylation factor; αSMA; α-Smooth muscle actin; Mkl1; Megakaryoblastic leukemia 1; MRTF-A; Myocardin-related transcription factor; sm22α; Smooth muscle 22α; VSMC; vascular smooth muscle cells; SecinH3; ADP-ribosylation factor (ARF); G-actin