
Ketones inhibit mitochondrial production of reactive oxygen species production following glutamate excitotoxicity by increasing NADH oxidation
Keywords: دی هیدروتیدیم; glutamate; neurotoxicity; diet; mitochondria; oxidation; stress; ACA; acetoacetate; BHB; β-hydroxybutyrate; DHE; dihydroethidium; EGTA; ethyleneglycol-bis-(β-aminoethyl ether)N,N,Nâ²,Nâ²-tetraacetic acid; FCCP; carbonyl cyanide p-[trifluoromethoxy]-ph