
PI3Kδ drives the pathogenesis of experimental autoimmune encephalomyelitis by inhibiting effector T cell apoptosis and promoting Th17 differentiation
Keywords: آنسفالومیلیت خودایمنی تجربی; Experimental autoimmune encephalomyelitis; Multiple sclerosis; Apoptosis; Phosphoinositide 3-kinase delta; Th17;