
Protective effects of dioscin against doxorubicin-induced nephrotoxicity via adjusting FXR-mediated oxidative stress and inflammation
Keywords: تومور نکروز عامل آلفا; Dox; doxorubicin; FXR; the farnesoid X receptor; AMPKα; AMP- activated protein kinase α; HO-1; hemeoxygenase-1; GST; glutathione-s-transferase; Nrf2; NF-E2-related factor 2; BUN; blood urea nitrogen; Cr; creatinine; HMGB1; high mobility group box 1; NF-