Keywords: میوکسی های قلبی; Action potentials; cardiac myocytes; cardiac output; haemodynamics; MRCP; sarcomeres; stroke volume;
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Keywords: میوکسی های قلبی; Cardiac myocytes; Reaction diffusion equation; Excess buffer; Finite element method
Keywords: میوکسی های قلبی; VLDL; Fatty acids; Insulin; Metabolic stress; Glucose transport; Cardiac myocytes
Keywords: میوکسی های قلبی; Cyclic nucleotide cross-talk signaling network; Phosphodiesterases; β-adrenergic pathway; NO/cGMP/PKG pathway; Cardiac myocytes; Computational model;
Keywords: میوکسی های قلبی; INa; voltage-gated sodium current; NaV; voltage-gated sodium channel; qPCR; quantity real-time polymerase chain reaction; RT-PCR; reverse transcription polymerase chain reaction; Cardiac myocytes; Voltage-gated sodium current; Distinctive properties
Keywords: میوکسی های قلبی; Ursolic acid; Heat stress; Apoptosis; Cardiac myocytes
Keywords: میوکسی های قلبی; Hypertrophy; Cardiac myocytes; Oxytocin; Endothelin 1; Atrial natriuretic peptide; Cyclic GMP;
Keywords: میوکسی های قلبی; Pulmonary hypertension; Microtubules; Cardiac myocytes; Right ventricle; Monocrotaline;
Keywords: میوکسی های قلبی; Cardiac myocytes; External magnetic fields; Magnetic nanoparticle drug delivery; Magnetic-field guided;
Stabilization of Ca2+ signaling in cardiac muscle by stimulation of SERCA
Keywords: میوکسی های قلبی; Cardiac myocytes; Calcium signaling; Sarcoplasmic reticulum calcium pump; Calcium waves; Arrhythmia; AP; Action Potential; β-AR; β-adrenergic receptor; CaMKII; Calmodulin kinase II; CICR; Ca2+-induced Ca2+ release; CPVT; Catecholaminergic polymorphic ve
A new interaction between proximal and distal C-terminus of Cav1.2 channels
Keywords: میوکسی های قلبی; Cav1.2; Cardiac myocytes; Calmodulin; Patch clamp; Pull-down assay;
(-)-Terpinen-4-ol changes intracellular Ca2+ handling and induces pacing disturbance in rat hearts
Keywords: میوکسی های قلبی; (-)-Terpinen-4-ol (PubChem CID: 5325830); (-)-Terpinen-4-ol; Heart; Arrhythmia; Cardiac myocytes; Ca2+ current; Alpinia zerumbet;
Subcellular localization of Na/K-ATPase isoforms in ventricular myocytes
Keywords: میوکسی های قلبی; Na/K ATPase; Na-pump; Cardiac myocytes; Transverse tubules; Ryanodine receptor; Super-resolution microscopy;
Mechanisms of the cyclic nucleotide cross-talk signaling network in cardiac L-type calcium channel regulation
Keywords: میوکسی های قلبی; Phosphodiesterases; Cyclic nucleotides; Signaling network; L-type calcium channel; Cardiac myocytes; Computational model;
Estrogen receptor profiling and activity in cardiac myocytes
Keywords: میوکسی های قلبی; Estrogen; Cardiac myocytes; Estrogen receptors; EstradiolERα, estrogen-receptor-alpha; E2, estrogen or 17β-estradiol; PI3K, phosphatidylinositide 3-kinase; MAPK, mitogen activated kinase; RVM, rat ventricular myocyte
p38α MAPK inhibits stretch-induced JNK activation in cardiac myocytes through MKP-1
Keywords: میوکسی های قلبی; Cardiac myocytes; p38; JNK; Mechanical stretch; MKP-1; PP1;
Protection of neonatal rat cardiac myocytes against radiation-induced damage with agonists of growth hormone-releasing hormone
Keywords: میوکسی های قلبی; Cardiac myocytes; Cardioprotection; GHRH agonists; GHRH/SV1 receptors; Radiation damage;
Testosterone protects cardiac myocytes from superoxide injury via NF-κB signalling pathways
Keywords: میوکسی های قلبی; Testosterone; Cardiac myocytes; Superoxide injury; Androgen receptor (AR); Nuclear factor-κB (NF-κB)Testosterone (PubChem CID 6013); Hydrogen peroxide (PubChem CID 784); Flutamide (PubChem CID 3397)
Cardiac CaMKIIδ splice variants exhibit target signaling specificity and confer sex-selective arrhythmogenic actions in the ischemic-reperfused heart
Keywords: میوکسی های قلبی; Cardiac myocytes; Ischemia/reperfusion; Ca2Â + regulation; Ventricular arrhythmia; Sex-specific;
Activation of PPARβ/δ protects cardiac myocytes from oxidative stress-induced apoptosis by suppressing generation of reactive oxygen/nitrogen species and expression of matrix metalloproteinases
Keywords: میوکسی های قلبی; Cardiac myocytes; Oxidative stress; Apoptosis; Matrix metalloproteinases; PPARβ/δ; Caspase 3; Bcl-2Hydrogen peroxide (PubChem CID: 784); GW0742 (PubChem CID: 9934458); GSK0660 (PubChem CID: 46233311)
Interaction between phosphodiesterases in the regulation of the cardiac β-adrenergic pathway
Keywords: میوکسی های قلبی; Phosphodiesterase; Cyclic nucleotides; β-adrenergic pathway; Signaling networks; Cardiac myocytes; Computational model
BNIP3 promotes calcium and calpain-dependent cell death
Keywords: میوکسی های قلبی; BNIP3; Calpain; Apoptosis; Cardiac myocytes; Caspases; Heart; Mitochondrial permeability transition pore; Calcium; Hypoxia; Acidosis; Ischemia
The lipid peroxidation product 4-hydroxy-trans-2-nonenal causes protein synthesis in cardiac myocytes via activated mTORC1-p70S6K-RPS6 signaling
Keywords: میوکسی های قلبی; 4-HNE; mTORC1; Protein synthesis; Cardiac myocytes; Erk1/2; Free radicals;
STIM1 elevation in the heart results in aberrant Ca2Â + handling and cardiomyopathy
Keywords: میوکسی های قلبی; AngII; angiotensin II; BDM; 2,3-butanedione monoxime; CaMKII; Ca2Â +/calmodulin-dependent protein kinase II; CMF; Ca2Â +- and Mg2Â +-free Ringer's solution; CPA; cyclopiazonic acid; EC; excitation-contraction; ER; endoplasmic reticulum; LTCC; L-type Ca2
Phenotypic screen quantifying differential regulation of cardiac myocyte hypertrophy identifies CITED4 regulation of myocyte elongation
Keywords: میوکسی های قلبی; Cardiac hypertrophy; Phenotypic screen; Cardiac myocytes; Signal transduction; CTGF; CITED4;
Synergistic induction of miR-126 by hypoxia and HDAC inhibitors in cardiac myocytes
Keywords: میوکسی های قلبی; Hypoxia; Cardiac myocytes; Micro-RNA-126; Tricostatin A; HDAC inhibitor;
NCLX: The mitochondrial sodium calcium exchanger
Keywords: میوکسی های قلبی; Mitochondria; Calcium transport; Cardiac myocytes; Sodium/calcium exchange; NCLX; Computational model;
T-type Ca2Â + channels regulate the exit of cardiac myocytes from the cell cycle after birth
Keywords: میوکسی های قلبی; T-type Ca2Â + channels; Cardiac myocytes; Cell cycle;
Protein-L-isoaspartate (D-aspartate) O-methyltransferase protects cardiomyocytes against hypoxia induced apoptosis through inhibiting proapoptotic kinase Mst1
Keywords: میوکسی های قلبی; Mst1 kinase; PCMT1; Hypoxia/reoxygenation; Cardiac myocytes; Apoptosis;
Components of the interleukin-33/ST2 system are differentially expressed and regulated in human cardiac cells and in cells of the cardiac vasculature
Keywords: میوکسی های قلبی; Interleukin-33; ST2; Cardiac fibroblasts; Cardiac myocytes; Cytokines;
Chymase mediates angiotensin-(1-12) metabolism in normal human hearts
Keywords: میوکسی های قلبی; ACE2; cardiac myocytes; angiotensin-converting enzyme inhibitors; angiotensin II; heart disease; proangiotensin 12; angiotensin-(1-7)
Mechanical stretch via transforming growth factor-β1 activates microRNA-208a to regulate hypertrophy in cultured rat cardiac myocytes
Keywords: میوکسی های قلبی; cardiac hypertrophy; cardiac myocytes; mechanical stretch; microRNA; transforming growth factor-β1
Trypanosoma cruzi infection induces morphological reorganization of the myocardium parenchyma and stroma, and modifies the mechanical properties of atrial and ventricular cardiomyocytes in rats
Keywords: میوکسی های قلبی; Cardiac myocytes; Chagas' disease; Pathology; Extracellular matrix; Oxidative stress; X-ray spectroscopy
Calmodulin binding proteins provide domains of local Ca2+ signaling in cardiac myocytes
Keywords: میوکسی های قلبی; Ca signaling; Calmodulin; Cardiac myocytes; Ion channels; Compartmentation;
Ca2+-activated adenylyl cyclase 1 introduces Ca2+-dependence to beta-adrenergic stimulation of HCN2 current
Keywords: میوکسی های قلبی; Hyperpolarization-activated channels; Cardiac myocytes; cAMP; Adenylyl cyclase; Adrenergic modulation;
Thermal dependence of cardiac SR Ca2+-ATPase from fish and mammals
Keywords: میوکسی های قلبی; PMSF, Phenylmethanesulfonyl fluoride; EGTA, Ethylene glycol-bis-(2-aminoethyl)-N,N,N′, N′-tetraacetic acidSERCA2; Cardiac myocytes; Cold adaptation; Endothermic fish; Ventricles
A new method of ultrasonic nonviral gene delivery to the adult myocardium
Keywords: میوکسی های قلبی; Ultrasound; Plasmid; siRNA; Liposomes; Gene transfer; Cardiac myocytes;
A natural compound induced cardiogenic differentiation of endogenous MSCs for repair of infarcted heart
Keywords: میوکسی های قلبی; Myocardial infarction; Mesenchymal stem cells; Cardiogenic differentiation; Myocardial regeneration; Cardiac myocytes
Focal adhesion kinase governs cardiac concentric hypertrophic growth by activating the AKT and mTOR pathways
Keywords: میوکسی های قلبی; FAK; focal adhesion kinase; FAK-Tg; cardiac-specific FAK transgenic mice model; FAKi; pharmacological FAK inhibitor (PF 573228); RP; rapamycin; TAC; thoracic aortic constriction; Cardiac myocytes; Cardiac failure; Mechanotransduction; Signal transduction;
Automated imaging reveals a concentration dependent delay in reversibility of cardiac myocyte hypertrophy
Keywords: میوکسی های قلبی; Cardiac hypertrophy; Automated imaging; α-Adrenergic signaling; Cardiac myocytes; Computational model
Isoforms of protein 4.1 are differentially distributed in heart muscle cells: Relation of 4.1R and 4.1G to components of the Ca2Â + homeostasis system
Keywords: میوکسی های قلبی; Cardiac myocytes; Sarcoplasmic reticulum; Intercalated disc; Cytoskeleton; Protein 4.1; Spectrin; SERCA2;
Cardiodepressive effect elicited by the essential oil of Alpinia speciosa is related to L-type Ca2+ current blockade
Keywords: میوکسی های قلبی; Alpinia speciosa; Essential oil; Cardiac myocytes; Rat atria; L-type Ca2+ channels;
Structure of the NH2-terminal variable region of cardiac troponin T determines its sensitivity to restrictive cleavage in pathophysiological adaptation
Keywords: میوکسی های قلبی; NH2-terminal variable region of troponin T; Restrictive proteolysis; Calcium overloading; Phosphorylation; Cardiac myocytes; Myocardial adaptation;
Monophosphothreonyl extracellular signal-regulated kinases 1 and 2 (ERK1/2) are formed endogenously in intact cardiac myocytes and are enzymically active
Keywords: میوکسی های قلبی; AdV; adenovirus or adenoviral; ET-1; endothelin-1; FPLC; fast protein liquid chromatography; MBP; myelin basic protein; MKK; MAPK kinase; PMA; phorbol 12-myristate 13-acetate; PTP; protein Tyr-phosphatase; SDS-PAGE; sodium dodecyl sulphate-polyacrylamide
Alpha-1-adrenergic receptors: Targets for agonist drugs to treat heart failure
Keywords: میوکسی های قلبی; alpha-1-Adrenergic receptors; Cardiac myocytes; Heart failure; Drug development
Regulation of cardiac Ca2+ channel by extracellular Na+
Keywords: میوکسی های قلبی; NCX, Na+/Ca2+ exchanger; CICR, Ca2+-induced Ca2+ release; CDI, Ca2+-dependent inactivation; PKA, protein kinase A; GFP, green fluorescent protein; SR, sarcoplasmic reticulum; I–V, current–voltageL-type Ca2+ channel; Na+-regulation; Cardiac myocytes; Ion c
Physiologic gating properties of unitary cardiac L-type Ca2+ channels
Keywords: میوکسی های قلبی; Unitary L-type Ca2+ channels; Gating kinetics; Excitation-contraction coupling; Cardiac myocytes; Single Ca2+ currents;
Enzyme electrodes to monitor glucose consumption of single cardiac myocytes in sub-nanoliter volumes
Keywords: میوکسی های قلبی; Glucose sensor; Glucose oxidase; Spin coating deposition; Microfluidic device; Cardiac myocytes
A novel secretagogue increases cardiac contractility by enhancement of L-type Ca2+ current
Keywords: میوکسی های قلبی; L-type Ca2+ current; Cardiac myocytes; Rabbit; Inotropy; N′1-(3,3,6,8-tetramethyl-1-oxo-1,2,3,4-tetrahydronaphthalen-2-yliden)-2-cyanoethanohydrazide
Angiotensin II inhibits the electrogenic Na+/HCO3â cotransport of cat cardiac myocytes
Keywords: میوکسی های قلبی; Na+/HCO3â co-transporter; Angiotensin II; Cardiac myocytes; p38 kinase; ERK 1/2 kinase;