
Regionally selective activation of ERK and JNK in morphine paradoxical hyperalgesia: A step toward improving opioid pain therapy
Keywords: MAPK; Morphine; Hyperalgesia; Protein kinase C; Pain; CREB; cyclic AMP response element-binding protein; DMSO; dimethyl sulfoxide; ERK; extracellular signal-regulated kinase; GFAP; glial fibrillary acidic protein; Iba1; ionized calcium binding adaptor mol