Lack of PPARα exacerbates lipopolysaccharide-induced liver toxicity through STAT1 inflammatory signaling and increased oxidative/nitrosative stress
Keywords: گیرنده های پولی مانند; LPS; lipopolysaccharide; TLR; Toll-like receptors; TNFα; tumor necrosis factor-α; NF-κB; nuclear factor-κB; MAPK; mitogen activated protein kinase; STAT; signal transducers and activators of transcription; ALT; alanine aminotransferase; WT; wild-type;