Nurr1 is not essential for the development of prepulse inhibition deficits induced by prenatal immune activation

► Prenatal immune challenge leads to post-pubertal onset of prepulse inhibition deficits and increased Nurr1 expression. ► Prenatal poly(I:C) treatment is similarly effective in disrupting prepulse inhibition in wild-type and Nurr1-deficient mice. ► Nurr1 is not essential for the development of prepulse inhibition deficits induced by prenatal immune activation.