کد مقاله | کد نشریه | سال انتشار | مقاله انگلیسی | نسخه تمام متن |
---|---|---|---|---|
6119354 | 1218755 | 2012 | 15 صفحه PDF | دانلود رایگان |
عنوان انگلیسی مقاله ISI
Immune complex-mediated co-ligation of the BCR with FcγRIIB results in homeostatic apoptosis of B cells involving Fas signalling that is defective in the MRL/Lpr model of systemic lupus erythematosus
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موضوعات مرتبط
علوم زیستی و بیوفناوری
ایمنی شناسی و میکروب شناسی
ایمونولوژی
پیش نمایش صفحه اول مقاله
![عکس صفحه اول مقاله: Immune complex-mediated co-ligation of the BCR with FcγRIIB results in homeostatic apoptosis of B cells involving Fas signalling that is defective in the MRL/Lpr model of systemic lupus erythematosus Immune complex-mediated co-ligation of the BCR with FcγRIIB results in homeostatic apoptosis of B cells involving Fas signalling that is defective in the MRL/Lpr model of systemic lupus erythematosus](/preview/png/6119354.png)
چکیده انگلیسی
⺠Apoptosis resulting from co-ligation of the BCR and FcγRIIB is caspase 8-dependent. ⺠Such apoptosis is associated with mitochondrial and lysosomal disruption. ⺠Apoptosis resulting from co-ligation of the BCR and FcγRIIB is associated with Fas signalling. ⺠Apoptosis resulting from co-ligation of the BCR and FcγRIIB is defective in the MRL/Lpr model of SLE.
ناشر
Database: Elsevier - ScienceDirect (ساینس دایرکت)
Journal: Journal of Autoimmunity - Volume 39, Issue 4, December 2012, Pages 332-346
Journal: Journal of Autoimmunity - Volume 39, Issue 4, December 2012, Pages 332-346
نویسندگان
Verica Paunovic, Natalie A. Carter, Theresa Thalhamer, Derek Blair, Brian Gordon, Erica Lacey, Alison M. Michie, Margaret M. Harnett,