
Research ArticleGlycogen synthase kinase 3 involvement in the excessive proinflammatory response to LPS in patients with decompensated cirrhosis
Keywords: آکت; Cirrhosis; Alcohol; Innate immunity; Lipopolysaccharides; AKT; Glycogen synthase kinase 3; TLR4; Toll-like receptor 4; LPS; lipopolysaccharides; TNF-α; tumor necrosis factor-α; IL; interleukin; GSK3; glycogen synthase kinase 3; PI3K; phosphatidylinosito