Dioxin-induced acute cardiac mitochondrial oxidative damage and increased activity of ATP-sensitive potassium channels in Wistar rats
Keywords: دی متیل سولفوکسید; Bioenergetics; Dioxin; mitoKATP; Mitochondria; ROS; TCDD; 5-HD; 5-hydroxydecanoic acid; ADP; adenosine diphosphate; AhR; aromatic hydrocarbon receptor; ATP; adenosine triphosphate; BSA; bovine serum albumin; ÎΨ; mitochondrial transmembrane electric pote