
Thalidomide represses inflammatory response and reduces radiculopathic pain by inhibiting IRAK-1 and NF-κB/p38/JNK signaling
Keywords: IVD; intervertebral disc disease; MMPs; metalloproteinases; IRAK-1; interleukin-1 receptor-associated kinase 1; iNOS; inducible nitric oxide synthase; Thalidomide; Inflammatory response; Interleukin-1β; Matrix metalloproteinase; Intervertebral disc cells