Sympathetic activity induced by naloxone-precipitated morphine withdrawal is blocked in genetically engineered mice lacking functional CRF1 receptor
Keywords: نورآدرنالین ; BNST; bed nucleus of the stria terminalis; BSA; bovine serum albumin; CeA; central amygdala; CRF; corticotropin-releasing factor; CRF1R; CRF receptor subtype; HPA; hypothalamic-pituitary-adrenocortical; HPLC; high-performance liquid chromatography; MH