
PELP1 oncogenic functions involve alternative splicing via PRMT6
Keywords: Alternative splicing; Breast cancer; Epigenetics; PELP1; PRMT6; BCAS2; breast cancer amplified sequence2; ChIP; chromatin immunoprecipitation; CDK; cell cycle dependent kinases; E2; Estrogen; ERα; estrogen receptor alpha; HER2; human epidermal growth fac