
Restoration of glucose metabolism in leptin-resistant mouse hearts after acute myocardial infarction through the activation of survival kinase pathways
Keywords: مواد واکنش پذیر اسید تیوباربیتوریک; AMPK; AMP activated protein kinase; ATP; adenosine triphosphate; CNTF; ciliary neurotrophic factor; FA; fatty acid; MDA; malondialdehyde; MI; myocardial infarction; ObR; leptin receptor; ObRKO; leptin receptor knockout; ObR+/+; oil treated ObRKO mice; ObR