Overexpression of OCT4 induced by modulation of histone marks plays crucial role in breast cancer progression
Keywords: سلولهای بنیادی سرطانی; CSC; cancer stem cell; ChIP; chromatin immunoprecipitation; DZNeP; deazaneplanocin A hydrochloride; DNMT; DNA methyltransferase; EZH2; enhancer of Zeste homolog 2; HDAC; histone deacetylase; H3K4me3; histone 3 lysine 4 trimethylation; H3K9acS10p; histone